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Steroids for sale in karachi Testosterone cypionate is a slow acting injectable ester of the primary male androgen testosterone. It binds to the androgen receptors, inducing the metabolism of androgens by its active metabolite trenbolone. Testosterone cypionate is the main male androgen for use in clinical practice since it is the most powerful drug currently available for the treatment of male hypogonadism. Testosterone cypionate has similar pharmacokinetics as testosterone, in that it can be taken with or without food. It is taken sublingually or orally in a dose of 6.8 to 8.9 mg/kg/day for treatment of early hypogonadism in hypogonadal men, or 8.6 to 8.9 mg/kg/day for treatment of hypogonadism in men who are either hypogonadal or testosterone deficient. There are two variations of testosterone cypionate (the two forms being 1-(2,3)octanoic acid-alpha-methyldopainate and 1-(4,5-dihydro-5-methyl-5H-pyrido[2,3]-1-oxo-3,5-dihydroxyphenyl)methyl-α-methyldopainate) that differ in the mechanism of action and potency of their androgenic action; the latter (the form 1-(2,3)octanoic acid-alpha-methyldopainate) contains no testosterone. Carcinogenesis, Mutagenesis and Impairment of Fertility Carcinogenesis Carcinogenicity may be studied using two methods, that of genetic or in vivo studies and that of animal models, all designed to provide information regarding the potential mutagenicity of a chemical compound (in contrast to in vitro tests). In the first methodology, the genetic mutagenicity of a chemical compound is considered one of the most important determinants of its safety (see Safety Assessment chapter for more information). In the second method, in vivo and in vitro mutagenicity as well as possible carcinogenicity of a chemical compound is considered as a function of the structure, the dose, and the route of administration and, in the latter case, of possible interaction of the compound with other drugs, carcinogens, or environmental factors (e.g., other chemicals used in a process). The overall risk of cancer from a chemical in humans may be determined by the dose-response relation between the concentration and cancer probability. A significant Related Article: